Michel Royeck
Affiliations: | DFG German Research Foundation |
Area:
Sodium Channels, Neurophysiology, EpilepsyGoogle:
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Publications
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Royeck M, Kelly T, Opitz T, et al. (2015) Downregulation of Spermine Augments Dendritic Persistent Sodium Currents and Synaptic Integration after Status Epilepticus. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 35: 15240-53 |
Chen S, Su H, Yue C, et al. (2011) An increase in persistent sodium current contributes to intrinsic neuronal bursting after status epilepticus. Journal of Neurophysiology. 105: 117-29 |
Wimmer VC, Reid CA, Mitchell S, et al. (2010) Axon initial segment dysfunction in a mouse model of genetic epilepsy with febrile seizures plus. The Journal of Clinical Investigation. 120: 2661-71 |
Uebachs M, Opitz T, Royeck M, et al. (2010) Efficacy loss of the anticonvulsant carbamazepine in mice lacking sodium channel beta subunits via paradoxical effects on persistent sodium currents. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 8489-501 |
Royeck M, Horstmann MT, Remy S, et al. (2008) Role of axonal NaV1.6 sodium channels in action potential initiation of CA1 pyramidal neurons. Journal of Neurophysiology. 100: 2361-80 |